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Andersen-Tawil syndrome (ATS1)–associated ventricular arrhythmias are initiated by frequent, hypokalemia-exacerbated, triggered activity. Previous ex vivo studies in drug-induced Andersen-Tawil syndrome (DI-ATS1) models have proposed that arrhythmia propensity in DI-ATS1 derives from cytosolic Ca 2+ ([Ca 2+ ] i ) accumulation leading to increased triggered activity.The purpose...
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